I am beginning to wonder of the true validity of Covid-19.
Has here been any healthy people die from it? Most of the accounts I’ve read are just a sore throat and much much less symptoms than regular flu.
Every death on the news states nothing about the person except they are dead and had a positive test. (Almost all 80 year olds) What were the underlying health issues?
How many false positives are there from a test that was rushed to market? All tests have false positives!
I’m not a conspiracy theorist, yet.
My 62 year old practicing physician friend told me that certain medicines for autoimmune diseases compromise the immune system making one susceptible to this virus. That begs the question, how many of those younger victims had an autoimmune ailment like arthritis and were on medication. There's a lot more to this puzzle than what is being released to the general public, read BIG PHARMA.
https://www.ncbi.nlm.nih.gov/books/NBK27143/
From the link: My take, if you're on a steroid, you are a target for this disease.
Current treatments for immunological disorders are nearly all empirical in origin, using
immunosuppressive drugs identified by screening large numbers of natural and synthetic compounds. The drugs currently used to suppress the
immune system can be divided into three categories: first, powerful anti-inflammatory drugs of the corticosteroid family such as prednisone; second, cytotoxic drugs such as azathioprine and cyclophosphamide; and third, fungal and bacterial derivatives, such as cyclosporin A,
FK506 (tacrolimus), and rapamycin (sirolimus), which inhibit signaling events within
T lymphocytes. These drugs are all very broad in their actions and inhibit protective functions of the immune system as well as harmful ones. Opportunistic infection is therefore a common complication of immuno-suppressive drug therapy. The ideal immunosuppressive agent would be one that targets the specific part of the
adaptive immune response that causes tissue injury. Paradoxically, antibodies themselves, by virtue of their exquisite
specificity, might offer the best possibility for the therapeutic inhibition of specific immune responses. We will also consider experimental approaches to controlling specific immune responses by manipulating the local cytokine environment or by manipulating
antigen so as to divert the response from a pathogenic pathway to an innocuous one. We have discussed in Chapters 12 and 13 how the pathological responses that cause allergy, autoimmunity, or
graft rejection can be prevented by innocuous, nonpathological T-cell responses.